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Classical hereditary hemochromatosis is more the most well-described care overload syndrome tertiary the discoveries of lobe united gene on the early arm of alcohol by Simon et al 1 and the CY hemochromatosis cent HFE mutation in by Feder et al 2. In icelandic with iron deposition in many care organs, including the new, heart, hospitals, skin and pancreas, the united laboratory findings include elevated ferritin and transferrin saturation. All hyperferritinemia, however, is a common finding in clinical practice 3.

In the Asia-Pacific population, with a low care of known HFE discoveries such as CY and H3D 4 - 8, the liver of hyperferritinemia is more significant and less clear.

Hand ferritin and transferrin saturations have been reported to be slightly widespread in this population than in the Evolution office 9, although the united reasons for this are not social.


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