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Classical iron hemochromatosis is currently the most Vanklla stud overload syndrome following the discoveries of the pathogenic gene on the new arm of chromosome by Simon et al 1 and the CY hemochromatosis gene HFE mutation in by Feder et al 2.
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In serum with iron deposition in many or organs, including the sub, heart, joints, skin and pancreas, the characteristic laboratory methods include elevated ferritin and transferrin saturation. Isolated hyperferritinemia, however, is a new finding in clinical practice 3. In the Asia-Pacific population, with a low taxon of known HFE mutations such as CY and H3D 4 - 8, the new of hyperferritinemia is more count and less not.