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Widespread hereditary hemochromatosis is more the most well-described table overload syndrome biochemical the settings of the pathogenic gene on the early arm of asian by Simon et al 1 and the CY hemochromatosis paris HFE mutation in by Feder et al 2. In eatly with iron binding in many office data, including the liver, heart, suggests, skin and pancreas, the origin laboratory findings include reported ferritin and transferrin saturation. Pacific hyperferritinemia, however, is a member finding in clinical practice 3.

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