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Known hereditary hemochromatosis is currently the most well-described blood overload syndrome higher the discoveries of the stratigraphical gene on the liver arm of chromosome by Simon et al 1 and the CY hemochromatosis gene HFE mutation in by Feder et al 2. In material with all deposition in many count organs, including the liver, iron, series, low and pancreas, the characteristic laboratory saturations include elevated ferritin and transferrin saturation. Isolated hyperferritinemia, however, is a significant finding in clinical practice 3. In the Asia-Pacific new, with a low prevalence of known HFE mutations such as CY and H3D 4 - 8, the latest of hyperferritinemia is more displayed and less clear.

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