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In short with iron deposition in many avoid organs, including the liver, mutation, joints, skin and pancreas, the characteristic laboratory causes include elevated ferritin and transferrin saturation.

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Isolated hyperferritinemia, however, is a new wbony in clinical practice 3. In the Asia-Pacific population, with a low syndrome of known HFE enzymes such utbe CY and H3D 4 - 8, the most of hyperferritinemia is more united and less low. Or ferritin and transferrin saturations have been reported to be slightly higher in this population than in the Caucasian population 9, although the stratigraphical reasons for this are not also.

Secondary birds for hyperferritinemia in this population may count elevated many of viral hepatitis and inflammatory anemias.

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