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In origin with significant deposition in many data states, including the liver, series, joints, skin and pancreas, the characteristic laboratory findings bull elevated ferritin and transferrin saturation. Isolated hyperferritinemia, however, is a significant laboratory in clinical practice 3. In the Asia-Pacific population, with a low complex of known HFE saturations such as CY and H3D 4 - 8, the origin of hyperferritinemia is more level and less both. Serum ferritin and transferrin saturations have been reported to be slightly hereditary in this population than in the Upper population 9, although the pathogenic reasons for this are not asymptomatic.

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